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The frequency of molecular detection of virulence genes encoding cytolysin A, high-pathogenicity island and cytolethal distending toxin of Escherichia coli in cases of sudden infant death syndrome does not differ from that in other infant deaths and healthy infants, by A. R. Highet, A. M. Berry, K. A. Bettelheim and P. N. Goldwater

Journal of Medical Microbiology vol. 58, part 3, pp. 285 - 289

Supplementary Table S1. Oligonucleotide primers used in this study. [PDF] (54 kb)

Supplementary Fig. S1. Sequence alignment of three clyA variants, Shiga toxin-producing E. coli clyA, enterotoxigenic E. coli clyA mutant and uropathogenic E. coli clyA-like gene.

Supplementary Fig. S2. PCR to demonstrate that primer pairs irp2-F, irp2-R and clyA-F, clyA-R could be duplexed in the same reaction to simultaneously detect irp2and clyA.

[PDF of both figures] (83 kb)







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